A possible suggestion of molecular basis of the phenomenon of cell memory implies that
the signals between cells, included in our model, can depend on a set of epigenetic informa-
tion, comprised by specific types and locations of cell surface molecules, distributed during
cell divisions according to a set of laws, preserving some of them in each cell (Morozova,
Shubin, 2012). Thus, the distribution of these molecules of the epigenetic code which has
rested on the membranes of the control cells after amputation, will influence a process of new
signals formation and distribution, as during the process of regeneration, so in the normal
development. Then this additional level of molecular information can be considered as the
epigenetic code for the target morphology of an organism.